Most heart attacks don’t start with a clogged artery! They start with a microscopic storm — a small, invisible wound inside the artery wall.

That storm begins decades before symptoms, sometimes before high school. By the time someone hits their forties or fifties, the real question isn’t whether atherosclerosis has begun…It’s how far it’s already gone.

Let’s zoom in — literally inside the artery wall — and trace this disease from its very first spark.
From the moment an ApoB-containing particle sneaks beneath the endothelium…
To the slow, simmering inflammation that follows…
To the day a plaque finally stops being silent.

In this episode of LipidCurious, we’ll uncover

1. Anatomy & The Birth of an Atherosclerotic Plaque
2. Why Some Arteries Are More Prone to Atherosclerosis
3. Seeing, Treating, and Reversing Atherosclerosis

You’ll walk away seeing cholesterol numbers in a completely different light — not as abstract labs, but as a daily pressure on the artery wall.

By the end, you’ll understand why atherosclerosis is the slow burn beneath the surface — invisible for years, yet capable of erupting into the events we fear most.

Bonus: The Visual Guide for this episode is waiting for you — click here. For the full collection of visuals across episodes, visit the Podcast page.

Download the Free LipidCurious Starter Kit here

Questions or feedback? Reach out at hello@lipidcurious.com

Disclaimer: This podcast is for educational purposes only. It is NOT medical advice.

LDL may get all the blame — but there’s a darker twin hiding in the shadows.
Same cholesterol core. Same ApoB backbone.
But with one sinister twist: a sticky tail called apolipoprotein(a).
That extra piece transforms an ordinary LDL particle into something far more dangerous.

Meet Lipoprotein(a) — or Lp(a) — one of the strongest, most under-recognized genetic drivers of premature cardiovascular disease.
It’s pro-atherogenic, pro-thrombotic, and pro-inflammatory — a triple threat that can quietly turn even the healthiest-looking lipid panel into a ticking time bomb.

In this episode of LipidCurious, we’ll uncover

1. What is Lp(a)?

2. How and when to test for it?

3. What to do when Lp(a) is high?

We’ll walk through genetics, testing strategy, and real-world management — plus a glimpse into the promising therapies now in late-phase trials.
By the end, you’ll understand why Lp(a) is the ghost in the lipid story — invisible on a standard panel, but capable of haunting even the fittest hearts.

Bonus: The Visual Guide for this episode is waiting for you — click here. For the full collection of visuals across episodes, visit the Podcast page.

Download the Free LipidCurious Starter Kit here

Questions or feedback? Reach out at hello@lipidcurious.com

Disclaimer: This podcast is for educational purposes only. It is NOT medical advice.

LDL-C is the number we’ve all been trained to chase. But what if the real culprit isn’t the cholesterol itself… but the number of particles carrying it?

Meet ApoB — the quiet truth-teller of lipidology.
It doesn’t care how perfect your LDL number looks. It counts every atherogenic particle capable of injuring the arterial wall — the true measure of risk hiding beneath the surface.

In this episode of LipidCurious, we’ll dig into what makes ApoB the stronger, sharper, and more honest marker of atherogenic burden:

1. Why ApoB is a better measure of atherogenic burden
2. How ApoB drives atherosclerosis
3. How ApoB fits into clinical practice

We’ll also explore practical takeaways — when to order ApoB, what cutoffs to use, and how to apply it when your LDL and ApoB numbers don’t agree.
By the end, you’ll see why every atherogenic particle counts — and why ApoB just might be the number that finally tells the truth about cardiovascular risk.

Bonus: The Visual Guide for this episode is waiting for you — click here. For the full collection of visuals across episodes, visit the Podcast page.

Download the Free LipidCurious Starter Kit here

Questions or feedback? Reach out at hello@lipidcurious.com

Disclaimer: This podcast is for educational purposes only. It is NOT medical advice.